Fig. 3: The loss of AGPAT2 does not induce ER stress.
From: AGPAT2 acts at the crossroads of lipid biosynthesis and DRP1-mediated ER morphogenesis
a WT and AGPAT2-KO MEFs were analyzed by Western blotting using antibodies against ER stress markers, including BiP, eIF2α, phospho-eIF2α, and CHOP, along with tubulin as a loading control. As a positive control, WT cells were treated with 10 µg/ml tunicamycin for 24 h to induce ER stress. b Quantification of band intensity. Mean ± SD (n = 3 experiments). Statistical analysis was performed using one-way ANOVA with post-hoc Tukey. Source data are provided as a Source Data file.
