Fig. 9: Proposed working model of hnRNPM in oocyte development.
From: hnRNPM cooperates with BCAS2 to modulate alternative splicing during oocyte development
hnRNPM cooperates with BCAS2 to regulate alternative splicing during oocyte development. The depletion of hnRNPM results in reduced protein levels of BCAS2. The phenotype of disrupted cytoskeletal organization and impaired meiotic maturation in Hnrnpm-deficient oocytes is likely due to aberrant splicing of key genes governing these processes. The oocyte diagram is a modified version of an original Servier Medical Art resource, licensed under a CC BY 4.0.
