Abstract
Endothelial dysfunction is a systemic disorder that triggers vascular alterations, characterised by a reduction in nitric oxide (NO) synthesis and/or a defective vasodilatory response. Trimethylamine-N-oxide (TMAO) is a gut microbiota-derived dietary metabolite, while tumour necrosis factor alpha (TNF-α) functions as a pro-inflammatory cytokine. Both are known to induce inflammation, metabolic modulations, and endothelial dysfunction, contributing to cardiometabolic diseases. Nevertheless, the comparative effects of TMAO and TNF-α on inflammation and metabolic modulations have yet to be investigated. Here, using bulk RNA-sequencing (RNA-seq), real-time quantitative polymerase chain reaction (RT-qPCR) and interleukins/chemokines multiplex assays, we demonstrate significantly higher levels of inflammation, with a stronger cytokine response and activation of type I and II interferons, following TNF-α treatment compared to TMAO treatment in human dermal microvascular endothelial cells (HMEC-1). In addition, TNF-α upregulates the disassembly of the extracellular matrix (ECM), while ECM-related genes and pathways are either not modulated or down-regulated after TMAO treatment. Intriguingly, TMAO specifically induces a shift in energy metabolism (upregulation of OXPHOS (oxidative phosphorylation)), while TNF-α rather modulates lipid metabolism. In conclusion, this study reveals common pathways, but also key differences in molecular processes activated by TNF-α versus TMAO. These data are essential for identifying the most suitable in vitro human cellular models to study inflammation, as well as to discover novel targeted therapeutics to alleviate cardiometabolic conditions.
Data availability
The datasets generated and/or analysed during the current study are available in the Gene Expression Omnibus (GEO) repository, GSE235204 and in Sequence Read Archive (SRA) under accession number PRJNA1233874.
Abbreviations
- ADAM:
-
A disintegrin and metalloproteinase
- CVDs:
-
Cardiovascular diseases
- CYP7A1:
-
Cholesterol 7α-hydroxylase
- COL:
-
Collagens
- COX:
-
Cyclooxygenase
- ECM:
-
Extracellular matrix
- FDR:
-
False discovery rate
- FBS:
-
Fetal bovine serum
- FLN:
-
Filamins
- FLT:
-
Fms-related tyrosine kinases
- GEO:
-
Gene expression omnibus
- GO BPs:
-
Gene ontology biological processes
- GSEA:
-
Gene set enrichment analysis
- GAPDH:
-
Glyceraldehyde-3-phosphate dehydrogenase
- HMEC-1:
-
Human microvascular endothelial cells
- HPKs:
-
Human primary keratinocytes
- HUVEC:
-
Human umbilical vein endothelial cells
- ITG:
-
Integrins
- KEGG:
-
Kyoto encyclopaedia of genes and genomes
- LAM:
-
Laminins
- mTNF:
-
Membrane bound form of TNF
- NO:
-
Nitric oxide
- NF-κB:
-
Nuclear factor kappa B
- ORA:
-
Over-representation analysis
- OXPHOS:
-
Oxidative phosphorylation
- PDGF:
-
Platelet-derived growth factors
- PCA:
-
Principal component analysis
- ROS:
-
Reactive oxygen species
- RT-qPCR:
-
Real-time quantitative polymerase chain reaction
- RNA-seq:
-
RNA-sequencing
- sTNF:
-
Soluble TNF
- SRA:
-
Sequence Read Archive
- SEM:
-
Standard error of the mean
- TACE:
-
TNF-α converting enzyme
- TMAO:
-
Trimethylamine-N-oxide
- TNF-α:
-
Tumour necrosis factor alpha
- UQCR:
-
Ubiquinol-cytochrome c reductase
- UPR:
-
Unfolded protein response
- V-ATPase:
-
Vacuolar H+-ATPase
- VST:
-
Variance stabilizing transformation
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Acknowledgements
MS was a recipient of the SUTD Ph.D. Scholarship. The authors thank Associate Professor Xiaogang Liu, Associate Professor Desmond Loke and Dr Christina Tan for their support and guidance in this study.
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The research was partially funded by SUTD Start-up Research Grant (SRG-SMT-2020–156), SUTD-ZJU Grant (ZJUVP2000102), SUTD Kickstarter Initiative (SKI_2021_02_05), and the Agency for Science, Technology and Research (A*STAR). The sponsors have no role in the study design, collection, analysis, and interpretation of data, or writing of the report and in the decision to submit the article for publication.
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Meyammai Shanmugham: Investigation, conceptualization, methodology, formal analysis, visualisation, writing- original draft preparationArun George Devasia: Methodology, formal analysis, writing- review & editingGokce Oguz: Methodology, formal analysis, visualisation, conceptualization, writing - review & editingAdaikalavan Ramasamy: Supervision, methodology, formal analysis, visualisation, conceptualization, writing - review & editingZen Zhi Yan Lim: InvestigationSophie Bellanger: Supervision, writing - review & editingChen Huei Leo: Supervision, Conceptualization, project administration, methodology, formal analysis, visualisation, funding acquisition, writing - review & editing.
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Shanmugham, M., Devasia, A.G., Oguz, G. et al. Comparative analysis of pathways induced by TMAO and TNF-α in human microvascular endothelial cells. Sci Rep (2026). https://doi.org/10.1038/s41598-026-46466-1
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DOI: https://doi.org/10.1038/s41598-026-46466-1