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Showing 1–16 of 16 results

Advanced filters: Author: Hongzheng Zhao Clear advanced filters

Atomic-scale mechanism unlocks thermal-stable high-κ performance in HfO₂ via coherent interfaces

Developing hafnium dioxide-based high-κ dielectrics via morphotropic phase boundaries remains challenging. This study proposes a thermal-stable high-κ strategy via coherent interface design and elucidates the atomic-scale enhancement mechanism.

Yihao Shen
Hongzheng Wang
Huaijin Zhang
ResearchOpen Access16 Jan 2026
Nature Communications

P: 1-14
Prethermalization by random multipolar driving on a 78-qubit processor

The existence of a long-lived, prethermal regime in many-body systems with tunable heating rates, driven by structured random protocols, is observed using a 78-qubit superconducting quantum processor.

Zheng-He Liu
Yu Liu
Heng Fan
ResearchOpen Access28 Jan 2026
Nature

P: 1-7
A benchmark of expert-level academic questions to assess AI capabilities

Humanity’s Last Exam, a multi-modal benchmark at the frontier of human knowledge, is designed to be an expert-level closed-ended academic benchmark with broad subject coverage.

Long Phan
Alice Gatti
Davide Scaramuzza
ResearchOpen Access28 Jan 2026
Nature

Volume: 649, P: 1139-1146
Experimental observation of a time rondeau crystal

Previous work on periodically driven many-body systems has demonstrated the formation of time crystals that break time-translation symmetry. Now, more general phases with partial temporal ordering have been realized.

Leo Joon Il Moon
Paul M. Schindler
Ashok Ajoy
ResearchOpen Access14 Oct 2025
Nature Physics

Volume: 21, P: 1813-1819
Publisher Correction: Non-invasive prenatal sequencing for multiple Mendelian monogenic disorders using circulating cell-free fetal DNA

Jinglan Zhang
Jianli Li
Christine M. Eng
Amendments and Corrections20 Feb 2019
Nature Medicine

Volume: 25, P: 701-702
Machine learning prediction of STEMI incidence with SHAP interpretation of environmental determinants

Hongxin Xu
Hongzheng Guan
Tao Song
ResearchOpen Access17 Nov 2025
Scientific Reports

Volume: 15, P: 1-13
Non-invasive prenatal sequencing for multiple Mendelian monogenic disorders using circulating cell-free fetal DNA

A non-invasive prenatal test utilizing cell-free DNA simultaneously detects mutations in 30 genes frequently associated with dominant monogenic diseases and demonstrates high accuracy in human clinical samples.

Jinglan Zhang
Jianli Li
Christine M. Eng
Research28 Jan 2019
Nature Medicine

Volume: 25, P: 439-447
A lightweight model design approach for few-shot malicious traffic classification

Ruonan Wang
Minhuan Huang
Liqiang He
ResearchOpen Access21 Oct 2024
Scientific Reports

Volume: 14, P: 1-15
Phenotypic expansion of CACNA1C-associated disorders to include isolated neurological manifestations

Lance H. Rodan
Rebecca C. Spillmann
Vandana Shashi
Research23 Jun 2021
Genetics in Medicine

Volume: 23, P: 1922-1932
Variants in PRKAR1B cause a neurodevelopmental disorder with autism spectrum disorder, apraxia, and insensitivity to pain

Felix Marbach
Georgi Stoyanov
Christian P. Schaaf
ResearchOpen Access08 Apr 2021
Genetics in Medicine

Volume: 23, P: 1465-1473
Heterozygous loss-of-function variants significantly expand the phenotypes associated with loss of GDF11

Thomas A. Ravenscroft
Jennifer B. Phillips
Hugo J. Bellen
Research10 Jun 2021
Genetics in Medicine

Volume: 23, P: 1889-1900
Commonalities across computational workflows for uncovering explanatory variants in undiagnosed cases

Shilpa Nadimpalli Kobren
Dustin Baldridge
Isaac S. Kohane
ResearchOpen Access12 Feb 2021
Genetics in Medicine

Volume: 23, P: 1075-1085
An autosomal dominant neurological disorder caused by de novo variants in FAR1 resulting in uncontrolled synthesis of ether lipids

Sacha Ferdinandusse
Kirsty McWalter
Frédéric M. Vaz
ResearchOpen Access26 Nov 2020
Genetics in Medicine

Volume: 23, P: 740-750
Clinical sites of the Undiagnosed Diseases Network: unique contributions to genomic medicine and science

Kelly Schoch
Cecilia Esteves
Vandana Shashi
Research23 Oct 2020
Genetics in Medicine

Volume: 23, P: 259-271
Correction: Phenotypic expansion of CACNA1C-associated disorders to include isolated neurological manifestations

Lance H. Rodan
Rebecca C. Spillmann
Vandana Shashi
Amendments and Corrections14 Sept 2021
Genetics in Medicine

Volume: 23, P: 2016
Correction to: An autosomal dominant neurological disorder caused by de novo variants in FAR1 resulting in uncontrolled synthesis of ether lipids

Sacha Ferdinandusse
Kirsty McWalter
Frédéric M. Vaz
Amendments and CorrectionsOpen Access19 Oct 2021
Genetics in Medicine

Volume: 23, P: 2467

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Atomic-scale mechanism unlocks thermal-stable high-κ performance in HfO₂ via coherent interfaces

Prethermalization by random multipolar driving on a 78-qubit processor

A benchmark of expert-level academic questions to assess AI capabilities

Experimental observation of a time rondeau crystal

Publisher Correction: Non-invasive prenatal sequencing for multiple Mendelian monogenic disorders using circulating cell-free fetal DNA

Machine learning prediction of STEMI incidence with SHAP interpretation of environmental determinants

Non-invasive prenatal sequencing for multiple Mendelian monogenic disorders using circulating cell-free fetal DNA

A lightweight model design approach for few-shot malicious traffic classification

Phenotypic expansion of CACNA1C-associated disorders to include isolated neurological manifestations

Variants in PRKAR1B cause a neurodevelopmental disorder with autism spectrum disorder, apraxia, and insensitivity to pain

Heterozygous loss-of-function variants significantly expand the phenotypes associated with loss of GDF11

Commonalities across computational workflows for uncovering explanatory variants in undiagnosed cases

An autosomal dominant neurological disorder caused by de novo variants in FAR1 resulting in uncontrolled synthesis of ether lipids

Clinical sites of the Undiagnosed Diseases Network: unique contributions to genomic medicine and science

Correction: Phenotypic expansion of CACNA1C-associated disorders to include isolated neurological manifestations

Correction to: An autosomal dominant neurological disorder caused by de novo variants in FAR1 resulting in uncontrolled synthesis of ether lipids

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