USP5 inhibition enables potential therapy for t(8;21) AML through ubiquitin-mediated AML1-ETO degradation in patient-derived xenografts

Journal:
Science Translational Medicine
Published:
DOI:
10.1126/scitranslmed.adt9100
Affiliations:
3
Authors:
15
Go to article
Institutions Authors Share
State Key Laboratory of Medicinal Chemical Biology (SKLMCB), China
13.500000
13.500000
 0.90
Cancer Hospital and Shenzhen Hospital, CAMS & PUMC, China
1.000000
 0.07
University of Nottingham Ningbo China (UNNC), China
0.500000
 0.03
Interested in Oncology and Carcinogenesis?